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Correlation of Professional Antigen-Presenting Tbet + CD11c + B Cells With Bone Destruction in Untreated Rheumatoid Arthritis.

Sarah McGrathKristoffer GrimstadKatrin ThorarinsdottirKristina ForslindDaniel GlinatsiMonica Leu AgeliiAlaitz AranburuTimothy SundellCharlotte A JonssonAlessandro CamponeschiAnna-Karin Hultgård EkwallAndreas TilevikInger GjertssonInga-Lill Mårtensson
Published in: Arthritis & rheumatology (Hoboken, N.J.) (2024)
MBCs contribute to the pathogenesis of RA by promoting bone destruction through antigen presentation, T cell activation, and Th17 polarization.
Keyphrases
  • rheumatoid arthritis
  • bone mineral density
  • disease activity
  • soft tissue
  • bone loss
  • case report
  • bone regeneration
  • ankylosing spondylitis
  • nk cells