Role of chemokine C-C motif ligand-1 in acute and chronic pulmonary inflammations.
Hiroyuki KishiMasamichi SatoYoko ShibataKento SatoSumito InoueShuichi AbeTomomi KimuraMichiko NishiwakiKeiko YamauchiTakako NemotoAkira IgarashiYoshikane TokairinOsamu NakajimaIsao KubotaPublished in: SpringerPlus (2016)
CCL1 overexpression in the lungs did not change the acute inflammatory response induced by LPS, but enhanced granuloma formation after mycobacterial treatment, possibly through enhancing endoplasmic reticulum stress.
Keyphrases
- endoplasmic reticulum stress
- inflammatory response
- liver failure
- drug induced
- liver injury
- respiratory failure
- induced apoptosis
- aortic dissection
- mycobacterium tuberculosis
- pulmonary hypertension
- lipopolysaccharide induced
- lps induced
- toll like receptor
- hepatitis b virus
- transcription factor
- intensive care unit
- extracorporeal membrane oxygenation
- combination therapy
- liver fibrosis
- smoking cessation