ATP2C2 and DYX1C1 are putative modulators of dyslexia-related MMR.

Bent MüllerGesa SchaadtJohannes BoltzeFrank Emmrichnull nullMichael A SkeideNicole E NeefIndra KraftJens BrauerAngela D FriedericiHolger KirstenArndt Wilcke

Published in: Brain and behavior (2017)

Our findings corroborate the late component of MMR as a potential endophenotype for dyslexia and support tripartite relationships between dyslexia-related SNPs, the late component of MMR and dyslexia.

Keyphrases

small molecule
genome wide