Association of maternal air pollution exposure and infant lung function is modified by genetic propensity to oxidative stress.
Dwan VilcinsWen Ray LeeCindy PhamSam TannerLuke D KnibbsDavid P BurgnerTamara L BlakeToby MansellAnne-Louise PonsonbyPeter D Slynull nullPublished in: medRxiv : the preprint server for health sciences (2023)
levels is associated with lower FRC and higher LCI in infants with a genetic propensity to oxidative stress. There was no relationship between maternal exposure to air pollution with maternal and cord blood inflammation or OS biomarkers.
Keyphrases
- air pollution
- lung function
- oxidative stress
- cord blood
- birth weight
- particulate matter
- pregnancy outcomes
- cystic fibrosis
- chronic obstructive pulmonary disease
- dna damage
- genome wide
- ischemia reperfusion injury
- diabetic rats
- induced apoptosis
- copy number
- weight gain
- gestational age
- pregnant women
- body mass index
- heat shock