Salbutamol-induced electrophysiological changes show no correlation with electrophysiological changes during hyperinsulinaemic-hypoglycaemic clamp in young people with Type 1 diabetes.

P NovodvorskyA BernjakE J RobinsonA IqbalI A MacdonaldRichard M JacquesJ L B MarquesP J SheridanSimon R Heller

Published in: Diabetic medicine : a journal of the British Diabetic Association (2018)

Both hypoglycaemia and salbutamol caused pro-arrhythmogenic electrophysiological changes in people with Type 1 diabetes but were not related in any given individual. Salbutamol does not appear useful in assessing an individual's electrophysiological response to hypoglycaemia.

Keyphrases

type diabetes
high glucose
drug induced
endothelial cells