Login / Signup

Chrysin Attenuates High Glucose-Induced BMSC Dysfunction via the Activation of the PI3K/AKT/Nrf2 Signaling Pathway.

Yu LiXimei Wang
Published in: Drug design, development and therapy (2022)
Chrysin can protect BMSCs from high glucose-induced oxidative stress via the activation of the PI3K/AKT/Nrf2 pathway, and promote bone regeneration in type 1 diabetic rats.
Keyphrases
  • high glucose
  • oxidative stress
  • diabetic rats
  • endothelial cells
  • bone regeneration
  • signaling pathway
  • induced apoptosis
  • hydrogen peroxide
  • pi k akt
  • epithelial mesenchymal transition
  • drug induced