Reactivation of herpesvirus type 6 and IgA/IgM-mediated responses to activin-A underpin long COVID, including affective symptoms and chronic fatigue syndrome.
Aristo VojdaniAbbas F AlmullaBo ZhouHussein Kadhem Al-HakeimMichael MaesPublished in: Acta neuropsychiatrica (2024)
Reactivation of HHV-6, SARS-CoV-2 persistence, and autoimmune reactions to activin-A combined with activated immune-oxidative pathways play a major role in the pathophysiology of long COVID as well as the severity of its affective symptoms and CFS.