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HDAC4 mutations cause diabetes and induce β-cell FoxO1 nuclear exclusion.

Maolian GongYong YuLei LiangDogus VuralliSebastian FroehlerPeter KuehnenPhilipp Du BoisJingjing ZhangAidi CaoYuantao LiuKhalid HussainJens FielitzShiqi JiaWei ChenKlemens Raile
Published in: Molecular genetics & genomic medicine (2019)
Mutations in HDAC4 disrupt the deacetylation of FoxO1, subsequently decrease the β-cell function including insulin secretion, resulting in diabetes.
Keyphrases
  • type diabetes
  • cardiovascular disease
  • glycemic control
  • transcription factor
  • histone deacetylase
  • signaling pathway
  • pi k akt
  • single cell
  • cell therapy
  • mesenchymal stem cells
  • cell proliferation
  • weight loss