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TRAPPC11 and GOSR2 mutations associate with hypoglycosylation of α-dystroglycan and muscular dystrophy.

Austin A LarsonPeter R BakerMiroslav P MilevCraig A PressRonald J SokolMary O CoxJacqueline K LekostajAaron A StenceAaron D BosslerJennifer M MuellerKeshika PrematilakeThierry Fotsing TadjoCharles A WilliamsMichael SacherSteven A Moore
Published in: Skeletal muscle (2018)
Recessive mutations in TRAPPC11 and GOSR2 are associated with congenital muscular dystrophy and hypoglycosylation of α-dystroglycan. This is the first report linking membrane trafficking proteins to dystroglycanopathy and suggests that these genes should be considered in the diagnostic evaluation of patients with congenital muscular dystrophy and dystroglycanopathy.
Keyphrases
  • muscular dystrophy
  • duchenne muscular dystrophy
  • genome wide
  • gene expression
  • autism spectrum disorder
  • transcription factor
  • genome wide analysis