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Erythrocytes from hereditary xerocytosis patients heterozygous for KCNN4 V282M exhibit increased spontaneous Gardos channel-like activity inhibited by senicapoc.

Alicia RiveraDavid H VandorpeBoris E ShmuklerDenis R GallagherChristopher C FikryFrans A KuypersCarlo BrugnaraL Michael SnyderSeth L Alper
Published in: American journal of hematology (2017)
Keyphrases
  • end stage renal disease
  • ejection fraction
  • newly diagnosed
  • chronic kidney disease
  • peritoneal dialysis
  • early onset
  • patient reported outcomes