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Multiple clinically relevant immunotherapies prolong the function of microencapsulated porcine islet xenografts in diabetic NOD mice without the use of anti-CD154 mAb.

Susan A SafleyGraham F BarberRobert W HoldcraftLawrence S GazdaStephanie DuncansonMark C PoznanskyAthanassios SambanisCollin J Weber
Published in: Xenotransplantation (2020)
Multiple different immunotherapies which specifically inhibit CD4+ T cells, modulate T-cell trafficking, or interfere with antigen presentation can substitute for anti-CD154 mAb to prolong encapsulated islet xenograft function in diabetic NOD mice.
Keyphrases
  • high fat diet induced
  • type diabetes
  • wound healing
  • monoclonal antibody
  • innate immune
  • insulin resistance
  • metabolic syndrome