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Downregulation of RIG-I mediated by ITGB3/c-SRC/STAT3 signaling confers resistance to interferon-α-induced apoptosis in tumor-repopulating cells of melanoma.

Yong LiYingqiu SongPindong LiMingxing LiHaizhou WangTao XuXiongjie YuYuandong YuYunYan TaiPing ChenXiaojun CaiXianhe WangLongchao XiangRui DengXiufang ZhangLiping GaoXuanbin WangJing LiuFengjun Cao
Published in: Journal for immunotherapy of cancer (2020)
In melanoma TRCs, ITGB3-c-SRC-STAT3 pathway caused RIG-I repression and then affect STAT1 activation to cause resistance to IFN-α-induced apoptosis. RIG-I is a prognostic marker in patients with melanoma. Combination of STAT3 inhibitor and IFN-α could enhance the efficacy of melanoma treatment. Our findings may provide a new concept of combinatorial treatment for future immunotherapy.
Keyphrases
  • induced apoptosis
  • endoplasmic reticulum stress
  • signaling pathway
  • oxidative stress
  • cell proliferation
  • dendritic cells
  • skin cancer
  • immune response
  • tyrosine kinase
  • combination therapy
  • current status