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Overexpression of DEC1 in the epithelium of OSF promotes mesenchymal transition via activating FAK/Akt signal axis.

Xin HuWeiming WangYue HuWenxin ChenCan WangLiudi YangTing MaoKun XiaAnjie MinHaofeng XiongTong Su
Published in: Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology (2022)
In the epithelium of OSF, overexpression of DEC1 induced activation of FAK/Akt signal axis, caused mesenchymal transition in epithelial cells, and may promote malignant transformation of OSF. Targeting DEC1 in OSF could be promising a new target for the diagnosis and treatment of this process.
Keyphrases
  • cell proliferation
  • signaling pathway
  • bone marrow
  • stem cells
  • transcription factor
  • cell migration
  • diabetic rats
  • oxidative stress
  • drug delivery