Calcium sensing and signaling are impaired in the lumbar spine of a rat model of congenital kyphosis.
Itsuki TakahashiYusuke WatanabeHiroyuki SonodaDaisuke TsunodaIzuki AmanoNoriyuki KoibuchiHaku IizukaNoriaki ShimokawaPublished in: European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society (2023)
These results indicate that adequate calcium signaling is extremely important for the regulation of normal bone formation and may also be a key factor for understanding the pathogenesis of congenital kyphosis.
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