Disruption of Trip11 in cranial neural crest cells is associated with increased ER and Golgi stress contributing to skull defects in mice.

Hiroyuki YamaguchiMatthew D MeyerLi HeYoshihiro Komatsu

Published in: Developmental dynamics : an official publication of the American Association of Anatomists (2022)

We have recently reported that the molecular complex of ciliary protein and GMAP210 is required for collagen trafficking. In this paper, we further characterized the important role of Trip11 being possibly involved in the regulation of ER and Golgi stress during skull development.

Keyphrases

endoplasmic reticulum
induced apoptosis
estrogen receptor
cell cycle arrest
stress induced
breast cancer cells
high fat diet induced
endoplasmic reticulum stress
type diabetes
cell death
amino acid
cell proliferation
skeletal muscle
pi k akt