Short-term exposure to ambient air pollutants may induce oxidative damage and pro-inflammatory roles, together with counter-regulatory anti-inflammatory response.
Keyphrases
- oxidative stress
- air pollution
- inflammatory response
- particulate matter
- heavy metals
- dna damage
- transcription factor
- lipopolysaccharide induced
- electronic health record
- diabetic rats
- induced apoptosis
- ischemia reperfusion injury
- toll like receptor
- immune response
- data analysis
- signaling pathway
- risk assessment
- artificial intelligence
- heat stress