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Epstein-Barr virus-encoded miR-BART5-5p upregulates PD-L1 through PIAS3/pSTAT3 modulation, worsening clinical outcomes of PD-L1-positive gastric carcinomas.

Chan Jin YoonMee Soo ChangDong Ha KimWon KimBo Kyung KooSung-Cheol YunSung Han KimYang Soo KimJun Hee Woo
Published in: Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association (2020)
Our findings imply that miR-BART5-5p directly targets PIAS3 and augments PD-L1 through miR-BART5/PIAS3/pSTAT3/PD-L1 axis control. This contributes to antiapoptosis, tumor cell proliferation, invasion and migration, as well as immune escape, furthering gastric carcinoma progression and worsening the clinical outcome, especially in the PD-L1(+) group of patients with EBV-associated gastric carcinomas. miR-BART5-5p may, therefore, be amenable to PD-1/PD-L1 immune checkpoint inhibitor therapy.
Keyphrases
  • cell proliferation
  • epstein barr virus
  • long non coding rna
  • long noncoding rna
  • diffuse large b cell lymphoma
  • cell cycle
  • stem cells
  • mesenchymal stem cells
  • signaling pathway
  • bone marrow
  • cell migration