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Dihydrotestosterone inhibits arylsulfatase B and Dickkopf Wnt signaling pathway inhibitor (DKK)-3 leading to enhanced Wnt signaling in prostate epithelium in response to stromal Wnt3A.

Sumit BhattacharyyaLeo FefermanJoanne K Tobacman
Published in: The Prostate (2019)
Androgen-induced declines in ARSB and DKK3 may contribute to prostate carcinogenesis by sustained activation of Wnt signaling in prostate epithelium in response to stromal Wnt3A.
Keyphrases
  • prostate cancer
  • benign prostatic hyperplasia
  • cell proliferation
  • stem cells
  • signaling pathway
  • bone marrow
  • pi k akt
  • diabetic rats
  • drug induced
  • oxidative stress
  • induced apoptosis
  • stress induced