CAMSAP3 depletion induces lung cancer cell senescence-associated phenotypes through extracellular signal-regulated kinase inactivation.

Onsurang WattanathamsanPaninee ChetprayoonNaphat ChantaravisootPiriya WongkongkathepPithi ChanvorachoteVarisa Pongrakhananon

Published in: Cancer medicine (2021)

This study is the first to report an intriguing role of CAMSAP3 in lung carcinoma cell senescence-associated phenotypes via the modulation of p-ERK/cyclin D1 signaling.

Keyphrases

dna damage
endothelial cells
single cell
signaling pathway
stress induced
cell cycle
cell proliferation
cell therapy
transcription factor
protein kinase
stem cells
pi k akt
mesenchymal stem cells
bone marrow