Endoplasmic reticulum stress-mediated ferroptosis in granulosa cells contributes to follicular dysfunction of polycystic ovary syndrome driven by hyperandrogenism.
Huimin DingYu XiangQi ZhuHonghui WuTianyue XuZichao HuangHongshan Ge
Published in: Reproductive biomedicine online (2024)
Ferroptosis plays a part in reproductive injury mediated by hyperandrogens associated with PCOS, and may be regulated by endoplasmic reticulum stress.