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Glucocorticoid-induced leucine zipper (GILZ) inhibits B cell activation in systemic lupus erythematosus.

Sarah A JonesAndrew E J TohDragana OdobasicMarie-Anne Virginie OudinQiang ChengJacinta P W LeeStefan J WhiteBrendan E RussSimona InfantinoAmanda LightDavid M TarlintonJames HarrisEric F Morand
Published in: Annals of the rheumatic diseases (2015)
Our findings demonstrated that GILZ is a non-redundant regulator of B cell activity, with important potential clinical implications in SLE.
Keyphrases
  • systemic lupus erythematosus
  • high glucose
  • diabetic rats
  • transcription factor
  • disease activity
  • drug induced
  • human health
  • endothelial cells
  • risk assessment
  • oxidative stress