Hyperbaric oxygen therapy prevents subarachnoid hemorrhage-induced apoptosis and impaired contractility of the rabbit bladder.
Ilker TinayOzgur CelikCagri Akin SekerciSelin CadirciOzge CevikBengusu OrogluGoksel SenerTufan TarcanPublished in: Neurourology and urodynamics (2020)
Subarachnoid hemorrhage stimulates apoptosis of the rabbit bladder and impairs the contractile response of the rabbit bladder to carbachol. HBOT creates a protective effect in rabbit bladder tissues and restores SAH-induced changes.
Keyphrases
- subarachnoid hemorrhage
- brain injury
- induced apoptosis
- endoplasmic reticulum stress
- spinal cord injury
- cerebral ischemia
- oxidative stress
- urinary tract
- signaling pathway
- diabetic rats
- gene expression
- skeletal muscle
- smooth muscle
- cell death
- cell cycle arrest
- cell proliferation
- drug induced
- blood brain barrier
- endothelial cells