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N-Acetylcysteine protects against intrauterine growth retardation-induced intestinal injury via restoring redox status and mitochondrial function in neonatal piglets.

Hao ZhangYue LiYueping ChenLili ZhangTian Wang
Published in: European journal of nutrition (2018)
Dietary supplementation of NAC shows promise for attenuating the early intestinal injury of young piglets with IUGR, probably through its antioxidant action to restore redox status and mitochondrial function.
Keyphrases
  • oxidative stress
  • diabetic rats
  • transcription factor
  • high glucose
  • big data
  • electron transfer