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Targeting Wnt-ß-Catenin-FOSL Signaling Ameliorates Right Ventricular Remodeling.

Sreenath Reddy NayakantiAleksandra FriedrichPoonam SarodeLeili JafariGiovanni MaroliMario BoehmAlice BourgeoisYann GrobsFatemeh KhassafiCarsten KuenneStefan GuentherSwati DabralJochen WilhelmAstrid WeissAstrid WietelmannBaktybek KojonazarovWiebke JanssenMario LoosoFrances S de ManSteeve ProvencherKhodr TelloHorst-Walter BirkSebastien BonnetSoni Savai PullamsettiRalph T SchermulySoni Savai Pullamsett
Published in: Circulation research (2023)
Wnt- β-Catenin-FOSL signaling is centrally involved in the hypertrophic RV response to increased afterload, offering novel targets for therapeutic interference with RV failure in pulmonary hypertension.
Keyphrases
  • cell proliferation
  • mycobacterium tuberculosis
  • pulmonary hypertension
  • stem cells
  • epithelial mesenchymal transition
  • drug delivery
  • mouse model
  • pulmonary arterial hypertension