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Peptide-based PROTAC degrader of FOXM1 suppresses cancer and decreases GLUT1 and PD-L1 expression.

Kun WangXiaoyong DaiAlbert YuChunyan FengKewei LiuLaiqiang Huang
Published in: Journal of experimental & clinical cancer research : CR (2022)
Our results indicate that biologically targeted degradation of FOXM1 is an attractive therapeutic strategy, and antagonist peptide-containing FOXM1-PROTACs as both degrader and inhibitor of FOXM1 could be developed as a safe and promising drug for FOXM1-overexpressed cancer therapy.
Keyphrases
  • cancer therapy
  • drug delivery
  • papillary thyroid
  • signaling pathway
  • squamous cell carcinoma
  • young adults
  • drug induced
  • childhood cancer