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SEMA6A/RhoA/YAP axis mediates tumor-stroma interactions and prevents response to dual BRAF/MEK inhibition in BRAF-mutant melanoma.

Rossella LoriaValentina LaquintanaStefano ScaleraRocco FraioliValentina CapraraItalia FalconeChiara BazzichettoMarta Di MartileLaura RosanòDonatella Del BufaloGianluca BossiIsabella SperdutiIrene TerrenatoPaolo ViscaSilvia SodduMichele MilellaGennaro CilibertoRita FalcioniVirginia FerraresiGiulia Bon
Published in: Journal of experimental & clinical cancer research : CR (2022)
Overall, our results indicate that SEMA6A contributes to microenvironment-coordinated evasion of melanoma cells from dual BRAF/MEK inhibition and it might be a good candidate predictor of short-term benefit from dual BRAF/MEK inhibition.
Keyphrases
  • wild type
  • metastatic colorectal cancer
  • pi k akt
  • stem cells
  • signaling pathway
  • basal cell carcinoma