Tlr2 deficiency does not limit the development of left ventricular hypertrophy in a model of transverse aortic constriction induced pressure overload.
Tippaporn BualeongSied KebirDorothea HofLina GoelzMathias GraeweStefan Felix EhrentrautPascal KnuefermannGeorg BaumgartenRainer MeyerHeidi EhrentrautPublished in: Journal of negative results in biomedicine (2016)
These data suggest that Tlr2 deficiency may promote the development of cardiac hypertrophy and ventricular remodeling after transverse aortic constriction.
Keyphrases
- left ventricular
- toll like receptor
- neuropathic pain
- heart failure
- inflammatory response
- aortic valve
- hypertrophic cardiomyopathy
- immune response
- cardiac resynchronization therapy
- pulmonary artery
- aortic stenosis
- mitral valve
- high glucose
- replacement therapy
- diabetic rats
- big data
- spinal cord injury
- coronary artery
- coronary artery disease
- endothelial cells
- spinal cord
- machine learning
- drug induced
- pulmonary hypertension
- artificial intelligence
- percutaneous coronary intervention
- ejection fraction