These data indicate that NADPH oxidase has a key role in facilitating NMDAR NR2A tyrosine phosphorylation via SFK activation during post-ischemic reperfusion.
Keyphrases
- cerebral ischemia
- protein kinase
- induced apoptosis
- acute myocardial infarction
- subarachnoid hemorrhage
- acute ischemic stroke
- cell cycle arrest
- high glucose
- blood brain barrier
- brain injury
- diabetic rats
- electronic health record
- blood glucose
- big data
- endoplasmic reticulum stress
- drug induced
- type diabetes
- oxidative stress
- ischemia reperfusion injury
- binding protein
- cell death
- adipose tissue
- percutaneous coronary intervention
- pi k akt
- coronary artery disease
- deep learning
- endothelial cells
- weight loss
- data analysis
- insulin resistance
- glycemic control
- stress induced
- cell proliferation