Iron chelation suppresses secondary bleeding after intracerebral hemorrhage in angiotensin II-infused mice.
Jie WangXiao-Qin TangMin XiaCheng-Cheng LiChao GuoHong-Fei GeYi YinBo WangWei-Xiang ChenHua FengPublished in: CNS neuroscience & therapeutics (2021)
Iron overload plays a key role in secondary bleeding after ICH in Ang II-induced hypertensive mice. Iron chelation during the process of Ang II-induced hypertension suppresses secondary bleeding after ICH.