Harnessing changes in open chromatin determined by ATAC-seq to generate insulin-responsive reporter constructs.
Collin B MerrillAustin B MontgomeryMiguel A PabonAndrey A ShabalinAylin R RodanAdrian RothenfluhPublished in: BMC genomics (2022)
Insulin-induced chromatin accessibility determined by ATAC-seq reveals enhancer regions that drive insulin-inducible reporter gene expression.
Keyphrases
- gene expression
- type diabetes
- genome wide
- transcription factor
- glycemic control
- dna methylation
- crispr cas
- dna damage
- single cell
- rna seq
- minimally invasive
- high glucose
- diabetic rats
- metabolic syndrome
- drug induced
- insulin resistance
- machine learning
- adipose tissue
- skeletal muscle
- oxidative stress
- artificial intelligence
- stress induced