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Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation.

Nicole TegtmeyerSteffen Backert
Published in: Disease models & mechanisms (2017)
Keyphrases
  • helicobacter pylori
  • helicobacter pylori infection
  • induced apoptosis
  • cell cycle arrest
  • endoplasmic reticulum stress
  • protein kinase
  • cell proliferation
  • pi k akt