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Genetic deletion of soluble epoxide hydrolase delays the progression of Alzheimer's disease.

Hsueh-Te LeeKuan-I LeeChia-Hui ChenTzong-Shyuan Lee
Published in: Journal of neuroinflammation (2019)
Our results suggest that sEH is a crucial regulator in the progression of AD and might be a potential therapeutic target for the treatment of AD.
Keyphrases
  • cognitive decline
  • transcription factor
  • genome wide
  • copy number
  • risk assessment
  • replacement therapy