Nicotine Potentially Alters Endothelial Inflammation and Cell Adhesion via LGALS9 .
Sönke Maximilian BraßAgnesa MazrekajJoscha MulorzWiebke IbingKim-Jürgen KrottKiku TakeuchiMelanie CappalloHsiang-Han LiuMargitta ElversHubert SchelzigMarkus Udo WagenhäuserPublished in: Journal of cardiovascular development and disease (2023)
itself may serve as key regulator for essential endothelial cell processes via interfering with various signaling pathways and may thus represent a potentially novel target in the pathogenesis of aortic pathologies.
Keyphrases
- cell adhesion
- endothelial cells
- signaling pathway
- oxidative stress
- aortic valve
- smoking cessation
- left ventricular
- transcription factor
- high glucose
- pulmonary artery
- epithelial mesenchymal transition
- aortic dissection
- induced apoptosis
- cell proliferation
- coronary artery
- pulmonary hypertension
- atrial fibrillation
- endoplasmic reticulum stress