SARS-CoV-2 infection of kidney tissues from severe COVID-19 patients.

Shawn RadovicWen MengLuping ChenAlberto E Paniz MondolfiClare BryceZachary GrimesEmilia M SordilloCarlos Cordon-CardoHaitao GuoYufei HuangShou-Jiang Gao

Published in: Journal of medical virology (2023)

In fatal COVID-19 kidneys, there are SARS-CoV-2 infection, minimally infiltrated innate immune cells, and evidence of viral replication, which could contribute to tissue damage in the form of ATN and glomerulosclerosis.

Keyphrases

sars cov
respiratory syndrome coronavirus
immune response
coronavirus disease
gene expression
oxidative stress
early onset
drug induced