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CCL3 contributes to secondary damage after spinal cord injury.

Nicolas PelischJose Rosas AlmanzaKyle E StehlikBrandy V AperiAntje Kroner
Published in: Journal of neuroinflammation (2020)
We have identified CCL3 as a potential target to modulate the inflammatory response and secondary damage after SCI. Collectively, this study shows that CCL3 contributes to progressive tissue damage and functional impairment during secondary injury after SCI.
Keyphrases
  • inflammatory response
  • liver fibrosis
  • oxidative stress
  • spinal cord injury
  • liver injury
  • drug induced
  • lipopolysaccharide induced
  • immune response
  • risk assessment
  • toll like receptor