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Analysis of autophagy in DLBCL reveals subtype-specific differences and the preferential targeting of ULK1 inhibition in GCB-DLBCL provides a rationale as a new therapeutic approach.

Harpreet K MandhairRamin RadpourMira WesterhuisYara BanzMagali HumbertMiroslav ArambasicJörn DengjelAndrew DaviesMario P TschanUrban Novak
Published in: Leukemia (2024)
Keyphrases
  • diffuse large b cell lymphoma
  • cell death
  • endoplasmic reticulum stress
  • signaling pathway
  • oxidative stress
  • cancer therapy
  • clinical trial
  • drug delivery