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The highly expressed calcium-insensitive synaptotagmin-11 and synaptotagmin-13 modulate insulin secretion.

Jones K OforiAlexandros KaragiannopoulosMohammad BarghouthMototsugu NagaoMarkus E AnderssonVishal A SalunkheEnming ZhangAnna WendtLena Eliasson
Published in: Acta physiologica (Oxford, England) (2022)
SYT11 and SYT13 have similar localization and transcriptional regulation, but they regulate insulin secretion differentially. While downregulation of SYT11 might be a compensatory mechanism in type-2 diabetes, downregulation of SYT13 reduces the insulin secretory response and overrules the compensatory regulation of SYT11 in a way that could aggravate the disease.
Keyphrases
  • type diabetes
  • cell proliferation
  • glycemic control
  • signaling pathway
  • cardiovascular disease
  • adipose tissue