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Rintatolimod in Advanced Pancreatic Cancer Enhances Antitumor Immunity through Dendritic Cell-Mediated T-Cell Responses.

Casper W F van EijckHassana El HaddaouiSongul KucukcelebiDisha VadgamaAmine FellahDana A M MustafaJoachim G J V AertsCasper H J van EijckMarcella Willemsen
Published in: Clinical cancer research : an official journal of the American Association for Cancer Research (2024)
This study presents compelling evidence of the immune-stimulatory properties linked to TLR-3 activation through rintatolimod. Rintatolimod may break immunologic tolerance by enhancing antitumor immunity through DC-mediated Th-cell responses. Furthermore, our findings lay the groundwork for investigating the potential synergy between TLR-3 activation and immune checkpoint inhibitor therapy to improve therapeutic outcomes. See related commentary by Martínez-Riaño et al., p. 3355.
Keyphrases
  • dendritic cells
  • toll like receptor
  • immune response
  • inflammatory response
  • single cell
  • cell therapy
  • regulatory t cells
  • type diabetes
  • stem cells
  • risk assessment
  • bone marrow
  • adipose tissue
  • skeletal muscle