Endothelial YAP/TEAD1-CXCL17 signaling recruits myeloid-derived suppressor cells against liver ischemia-reperfusion injury.

Sitong ZhangZhongquan SunZhenhua ChenYanli BiShenyu WeiZhengwei MaoJin JinYuan DingWei-Lin Wang

Published in: Hepatology (Baltimore, Md.) (2024)

Endothelial YAP/TEAD1-CXCL17 signaling recruited MDSCs to attenuate liver IRI, providing evidence of therapeutic potential for managing IRI in liver surgery.

Keyphrases

ischemia reperfusion injury
endothelial cells
induced apoptosis
minimally invasive
oxidative stress
cell cycle arrest
coronary artery disease
cell proliferation
signaling pathway
endoplasmic reticulum stress
atrial fibrillation
surgical site infection
pi k akt