ULK1 phosphorylates Sec23A and mediates autophagy-induced inhibition of ER-to-Golgi traffic.
Wenjia GanCaiyun ZhangKa Yu SiuAyano SatohJulian A TannerSidney Sb YuPublished in: BMC cell biology (2017)
These findings identify Sec23A as a new target of ULK1 and uncover a mechanism of coordinating intracellular protein transport and autophagy.