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ATM Regulates Differentiation of Myofibroblastic Cancer-Associated Fibroblasts and Can Be Targeted to Overcome Immunotherapy Resistance.

Massimiliano MelloneKlaudia PiotrowskaGiulia VenturiLija JamesAleksandra BzuraMaria A LopezSonya JamesChuan WangMatthew J EllisChristopher J HanleyJosephine F BuckinghamKerry L CoxGareth HughesViia Valge-ArcherEmma V KingStephen A BeersVincent JaquetGeorge D D JonesNatalia SavelyevaA Emre SayanJason L ParsonsStephen T DurantGareth J Thomas
Published in: Cancer research (2022)
ATM signaling supports the differentiation of myoCAFs to suppress T-cell infiltration and antitumor immunity, supporting the potential clinical use of ATM inhibitors in combination with checkpoint inhibition in myoCAF-rich, immune-cold tumors.
Keyphrases
  • dna damage
  • dna repair
  • dna damage response
  • oxidative stress
  • cell cycle