Overall, our findings reveal the detrimental role of USP38 in diabetes-related AF, manifested by increased level of iron overload and oxidation stress.
Keyphrases
- atrial fibrillation
- type diabetes
- iron deficiency
- cardiovascular disease
- catheter ablation
- left atrial
- oral anticoagulants
- left atrial appendage
- genome wide
- transcription factor
- heart failure
- glycemic control
- small molecule
- hydrogen peroxide
- protein protein
- binding protein
- metabolic syndrome
- gene expression
- stress induced
- skeletal muscle
- drug induced
- coronary artery disease
- mitral valve
- heat stress