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Synaptic retrograde regulation of the PKA-induced SNAP-25 and Synapsin-1 phosphorylation.

Aleksandra PolishchukVíctor Cilleros-MañéLaia Just-BorràsMarta Balanyà SeguraGenís Vandellòs PontCarolina Silvera SimónMarta TomàsNeus GarciaJosep Maria TomàsMaria Angel Lanuza
Published in: Cellular & molecular biology letters (2023)
This provides a molecular mechanism of the bidirectional communication between nerve terminals and muscle cells to balance the accurate process of ACh release, which could be important to characterize molecules as a therapy for neuromuscular diseases in which neuromuscular crosstalk is impaired.
Keyphrases
  • induced apoptosis
  • cell cycle arrest
  • high glucose
  • skeletal muscle
  • diabetic rats
  • oxidative stress
  • endoplasmic reticulum stress
  • cell death
  • peripheral nerve
  • mass spectrometry
  • prefrontal cortex