Bushen-Yizhi formula ameliorates mitochondrial dysfunction and oxidative stress via AMPK/Sirt1 signaling pathway in D-gal-induced aging rats.
Yanfang LiaoYiyi LaiHuilin XuLi GaoXiaomei FuXue WangQi WangJiangang ShenJiansong FangShuhuan FangPublished in: Chinese medicine (2023)
BSYZ treatment effectively rescues neurobehavioral impairment by improving mitochondrial dysfunction, oxidative stress, neuroinflammation and neuroapoptosis via AMPK/SIRT1 pathway in D-gal-induced aging.
Keyphrases
- oxidative stress
- diabetic rats
- ischemia reperfusion injury
- signaling pathway
- high glucose
- induced apoptosis
- dna damage
- skeletal muscle
- traumatic brain injury
- epithelial mesenchymal transition
- protein kinase
- lps induced
- endothelial cells
- preterm infants
- lipopolysaccharide induced
- cognitive impairment
- cell proliferation
- cerebral ischemia
- stress induced
- subarachnoid hemorrhage