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SARS-CoV-2 infection of kidney tissues from severe COVID-19 patients.

Shawn RadovicWen MengLuping ChenAlberto E Paniz MondolfiClare BryceZachary GrimesEmilia M SordilloCarlos Cordon-CardoHaitao GuoYufei HuangShou-Jiang Gao
Published in: Journal of medical virology (2023)
In fatal COVID-19 kidneys, there are SARS-CoV-2 infection, minimally infiltrated innate immune cells, and evidence of viral replication, which could contribute to tissue damage in the form of ATN and glomerulosclerosis.
Keyphrases
  • sars cov
  • respiratory syndrome coronavirus
  • immune response
  • coronavirus disease
  • gene expression
  • oxidative stress
  • early onset