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T Lymphocyte-Derived Exosomes Transport MEK1/2 and ERK1/2 and Induce NOX4-Dependent Oxidative Stress in Cardiac Microvascular Endothelial Cells.

Filip RolskiMarcin CzepielKarolina TkaczKatarzyna FrytMaciej SiedlarGabriela KaniaPrzemysław Błyszczuk
Published in: Oxidative medicine and cellular longevity (2022)
T cells in immune response and represent potential important triggers of NOX4-dependent endothelial dysfunction. Neutralization of the prooxidative aspect of CD4-exosomes could open perspectives for the development of new therapeutic strategies in inflammatory cardiovascular diseases.
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