Cognitive Training as a Potential Activator of Hippocampal Neurogenesis in the Rat Model of Sporadic Alzheimer's Disease.
Alena O BurnyashevaTatiana A KozlovaNatalia A StefanovaNataliya G KolosovaEkaterina A RudnitskayaPublished in: International journal of molecular sciences (2020)
There is a growing body of evidence that interventions like cognitive training or exercises prior to the manifestation of Alzheimer's disease (AD) symptoms may decelerate cognitive decline. Nonetheless, evidence of prevention or a delay of dementia is still insufficient. Using OXYS rats as a suitable model of sporadic AD and Wistar rats as a control, we examined effects of cognitive training in the Morris water maze on neurogenesis in the dentate gyrus in presymptomatic (young rats) and symptomatic (adult rats) periods of development of AD signs. Four weeks after the cognitive training, we immunohistochemically estimated densities of quiescent and amplifying neuronal progenitors, neuronal-lineage cells (neuroblasts and immature and mature neurons), and astrocytes in young and adult rats, and the amyloid precursor protein and amyloid-β in adult rats. Reference memory was defective in OXYS rats. The cognitive training did not affect neuronal-lineage cells' density in either rat strain either at the young or adult age, but activated neuronal progenitors in young rats and increased astrocyte density and downregulated amyloid-β in adult OXYS rats. Thus, to activate adult neurogenesis, cognitive training should be started before first neurodegenerative changes, whereas cognitive training accompanying amyloid-β accumulation affects only astrocytic support.
Keyphrases
- cognitive decline
- virtual reality
- cerebral ischemia
- mild cognitive impairment
- induced apoptosis
- middle aged
- young adults
- late onset
- cell cycle arrest
- blood brain barrier
- spinal cord injury
- cell death
- inflammatory response
- amyotrophic lateral sclerosis
- working memory
- human health
- binding protein
- pi k akt
- protein protein