Assessment of lactate production as a response to sustained intrapartum hypoxia in large-for-gestational-age newborns.

Mehreen Zaigham Karin Källen Per Olofsson

Published in: Acta obstetricia et gynecologica Scandinavica (2018)

Considering cord acidemia a proxy for intrapartum hypoxia, LGA fetuses showed no impaired ability to produce lactate during hypoxia. Maternal diabetes did not hamper the ability of LGA fetuses to produce lactate during hypoxia.

Keyphrases

gestational age
birth weight
preterm birth
endothelial cells
type diabetes
cardiovascular disease
low birth weight
preterm infants
glycemic control
adipose tissue
body mass index