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ER stress arm XBP1s plays a pivotal role in proteasome inhibition-induced bone formation.

Dan ZhangKim De VeirmanRong FanQiang JianYuchen ZhangLi LeiHolly EvansYanmeng WangLei LeiBaiyan WangRamone A WilliamsonAndrew ChantryPengcheng HeAng LiHendrik De RaeveKarin VanderkerkenAili HeJinsong Hu
Published in: Stem cell research & therapy (2020)
These results demonstrated that XBP1s is a master regulator of PI-induced osteoblast differentiation. Activation of IRE1α-XBP1s ER stress signaling can promote osteogenesis, thus providing a novel strategy for the treatment of myeloma bone disease.
Keyphrases
  • high glucose
  • diabetic rats
  • drug induced
  • endoplasmic reticulum stress
  • oxidative stress
  • transcription factor
  • endothelial cells
  • bone mineral density
  • multiple myeloma
  • newly diagnosed
  • soft tissue