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MAML1: a coregulator that alters endometrial epithelial cell adhesive capacity.

Sadaf ZafirWei ZhouEllen MenkhorstLeilani SantosEvdokia Dimitriadis
Published in: Fertility research and practice (2021)
Our data strongly suggest that MAML1 is involved in regulating the endometrial adhesive capacity and may facilitate embryo attachment, either directly or indirectly through the Notch signaling pathway.
Keyphrases
  • signaling pathway
  • endometrial cancer
  • pi k akt
  • electronic health record
  • epithelial mesenchymal transition
  • cell proliferation
  • oxidative stress
  • pregnancy outcomes
  • pregnant women
  • endoplasmic reticulum stress